School of Oral Health Sciences
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Item Intercellular junctions in the attached gingiva and alveolar mucosa in the vervet monkey (cercopithecus aethiops)(1979) Grossman, E. S.; Austin, J. C.; Cleaton-Jones, P.This study was undertaken to determine the distribution of hemidesmosomes on the basal cell membranes adjacent to the basement lamina and the spinous cell membranes on unloaded keratinized AG and non- keratinized AM.Item Primary fixation of vervet monkey oral epithelium for SEM and TEM(1987) Grossman, E. S.Vervet monkey attached gingiva and alveolar mucosa was used to investigate the effect of primary fixative composition and osmolarity on the scanning electron microscope appearance and epithelial cell surface feature density. Primary fixation was obtained using 12 different fixatives with osmolarities varying between 320-2010mOsm followed by further standard SEM processing procedures. All primary fixatives investigated produced acceptably fixed oral epithelium for SEM study, showing all the morphologic features characteristic of either keratinized or non-keratinized oral tissue. Point counting revealed that the density of microvilli of attached gingiva epithelial cells when fixed at 2010mOsm was 72 +/- 8% of the cell surface area. This decreased to 40 +/- 5% when fixed at 320mOsm. Similarly the microplication density of the alveolar mucosa epithelial cells decreased from 70 +/- 5% at 2010mOsm to 43 +/- 7% at 320mOsm. Both these differences proved to be highly significantItem Hard tissue barrier formation in pulpotomized monkey teeth capped with cyanoacrylate or calcium hydroxide for 10 and 60 minutes(1987) Cvek, M.; Granath, L.; Cleaton-Jones, P.; Et alMonkey incisor teeth were pulpotomized in groups of 10. After physiological hemostasis, the pulps of group I were covered with isobutyl cyanoacrylate, and those of groups II and III with calcium hydroxide for 10 and 60 minutes, respectively, whereafter this compound was washed away and the wound surfaces covered with Teflon. In group IV, calcium hydroxide was used as a positive control, and Teflon as a negative control in group V. The animals were killed after 12 weeks and the teeth removed in tissue blocks. The material was processed and evaluated histologically with respect to location and continuity of a hard tissue barrier, type of newly formed hard tissue, state of the pulp, and presence of stainable bacteria in the coronal cavity. Seven of nine teeth in group I showed a hard tissue barrier. The corresponding figure for group II was eight out of 10 teeth. All teeth in groups III and IV had a barrier. The incidence of a continuous barrier increased from group I through group IV, as did the incidence of its location below the level of the original wound surface. The condition of the pulp was related to the presence of bacteria and the continuity of the barrier to the presence of inflammation. There was no bridging in group V. The results support the theory that a low-grade irritation is responsible for the formation of a hard tissue barrier in exposed pulps.Item A proposed function for microplications on epithelial cells(1983) Grossman, E. S.Item A comparative study of the cellular response of the oral mucosa to mechanical loading(1981) Grossman, E. S.; Austin, J. C.Item Gingival temperatures in the baboon and Vervet Monkey(1990) Volchansky, A.; Cleaton-Jones, P.; Smith, M.Item Effect of specimen block orientation on separation artefact in pulpal sections(1988) van Wyk, J. H.Item Pulpal responses to two luting cements(1982) Valcke, C. F.; Cleaton-Jones, P. E.; Austin, J. C.; et al.Item A metrical study of some periodontal structures in the Vervet Monkey(1973) Volchansky, A.; Cleaton-Jones, P.Item The response of the exposed dental pulp to a calcium hydroxide-corticosteroid mixture(1976) Bloch, W. W.; Austin, J. C.