SARSCoV2 BA45 infection triggers more crossreactive FcRIIIa signalling and neutralization than BA1 in the context of hybrid immunity

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dc.article.end-page14en
dc.article.start-page1en
dc.citation.doi10.1128/JVI.00678-24en
dc.contributor.authorSimone Richardsonen
dc.contributor.authorNkululeko Mzindleen
dc.contributor.authorT Motlouen
dc.contributor.authorNelia Manamelaen
dc.contributor.authorM van der Meschten
dc.contributor.authorBronwen Lambsonen
dc.contributor.authorJosie Everatten
dc.contributor.authorD Amoakoen
dc.contributor.authorE "et al"en
dc.contributor.authorAnne Von Gottbergen
dc.contributor.authorNicole Wolteren
dc.contributor.authorJinal Bhimanen
dc.contributor.authorPenelope Mooreen
dc.contributor.authorNelia Manamelaen
dc.date.accessioned2024-11-27T12:33:08Z
dc.date.available2024-11-27T12:33:08Z
dc.facultyFACULTY OF HEALTH SCIENCESen
dc.identifier.citationWOSen
dc.identifier.issn0022-538Xen
dc.identifier.urihttps://hdl.handle.net/10539/42939
dc.journal.titleSARSCoV2 BA45 infection triggers more crossreactive FcRIIIa signalling and neutralization than BA1 in the context of hybrid immunityen
dc.journal.volume98en
dc.publisherAMER SOC MICROBIOLOGYen
dc.titleSARSCoV2 BA45 infection triggers more crossreactive FcRIIIa signalling and neutralization than BA1 in the context of hybrid immunityen
dc.typeJournal Articleen

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