The novel activity of carbamazepine as an activation modulator extends from NaV 1.7 mutations to the NaV 1.8-S242T mutant channel from a patient with painful diabetic neuropathy
dc.citation.doi | 10.1124/MOL.118.113076 | |
dc.citation.epage | 1269 | |
dc.citation.spage | 1256 | |
dc.contributor.author | C Han | |
dc.contributor.author | Andreas Themistocleous | |
dc.contributor.author | M Estacion | |
dc.contributor.author | F Dib-Hajj | |
dc.contributor.author | I Blesneac | |
dc.contributor.author | E et al | |
dc.date.accessioned | 2023-04-13T11:08:27Z | |
dc.date.available | 2023-04-13T11:08:27Z | |
dc.identifier.citation | ISI | |
dc.identifier.issn | 0026-895X | |
dc.identifier.uri | https://hdl.handle.net/10539/35084 | |
dc.journal.title | MOLECULAR PHARMACOLOGY | |
dc.journal.volume | 94 | |
dc.title | The novel activity of carbamazepine as an activation modulator extends from NaV 1.7 mutations to the NaV 1.8-S242T mutant channel from a patient with painful diabetic neuropathy |
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